The primary effects of cannabinoids are exerted in the central nervous system and expressed by a decreased motivation for physical effort, impairment of psychomotor performance as well as short-term memory and perception. In addition to psychological effects marijuana induces tachycardia, bronchodilation and an increased blood flow to the limbs. In users with some cardiovascular diseases (hypertension or atherosclerosis) the increased heart rate after cannabinoids consumption would have a serious medical adverse effect. Smoking of hashish or marijuana can induce the development of bronchitis and lung pathology.
Chronic consumption of cannabinoids could lead to destruction of CB1 receptors in the brain resulting in reduced blood flow and decreased glucose and oxygen supply to the brain tissues. The main results are attention deficits, memory loss, and impaired learning ability.
The various effects of cannabinoids on the central nervous system include:
Acute effects of hashish and marijuana are low with negligible risk of lethal intoxication. Addiction to the cannabinoids may be related to the dose applied.
Chronic consumption of cannabinoids could lead to destruction of CB1 receptors in the brain resulting in reduced blood flow and decreased glucose and oxygen supply to the brain tissues. The main results are attention deficits, memory loss, and impaired learning ability.
The various effects of cannabinoids on the central nervous system include:
- euphoria,
- panic attacks,
- impaired comprehension,
- altered sense of time,
- paranoia,
- anxiety,
- altered cognition,
- impaired learning,
- memory,
- perception,
- judgement,
- depersonalization,
- confusion,
- amnesia,
- and hallucinations.
Acute effects of hashish and marijuana are low with negligible risk of lethal intoxication. Addiction to the cannabinoids may be related to the dose applied.
Crosstalk:
Cannabinoids
Cannabinoids (hashish and marijuana) are substances able to elicit psychic changes like those manifested in the course of psychosis. Therefore, these substances are also called psychotomimetics, or psychodelics, or hallucinogens.
When cannabinoids are introduced into the body, its active ingredient, delta-9-tetrahydrocannabinol (THC) modifies the activity of the brain. THC acts on cannabinoid receptors bringing sensation of euphoria, relaxation and amplified sensory perception. The information concerning the presence of THC in the brain has been transmitted by an endogenous molecule – anandamide. Anandamide is involved in regulation of mood, appetite, pain, cognition and emotions. THC increases dopamine released by compensation of inhibitory effect of GABA neurons. Chronic consumption of cannabinoids could lead to destruction of some neuron receptors in the brain resulting in attention deficits, memory loss, impaired learning ability and psychomotor performance in the user. The growing number of young athletes using cannabinoids despite of the discipline of sport suggests that cannabinoids are not used for doping purpose but rather for social reasons.
When cannabinoids are introduced into the body, its active ingredient, delta-9-tetrahydrocannabinol (THC) modifies the activity of the brain. THC acts on cannabinoid receptors bringing sensation of euphoria, relaxation and amplified sensory perception. The information concerning the presence of THC in the brain has been transmitted by an endogenous molecule – anandamide. Anandamide is involved in regulation of mood, appetite, pain, cognition and emotions. THC increases dopamine released by compensation of inhibitory effect of GABA neurons. Chronic consumption of cannabinoids could lead to destruction of some neuron receptors in the brain resulting in attention deficits, memory loss, impaired learning ability and psychomotor performance in the user. The growing number of young athletes using cannabinoids despite of the discipline of sport suggests that cannabinoids are not used for doping purpose but rather for social reasons.
